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The findings of the study revealed that hyperoxia raised the initial amount of Nrf2 and many transcriptional targets including Immunoproteasome, 20S Proteasome, NQO1, Lon protease, and HO-1, but caused the loss of cellular capacity to adapt to signaling levels of H 2 O 2

10.1186/s12935-022-02679-8 141 MarketM.TennakoonG.ScaffidiM.CookD

Fixed modification was set to carbamidomethylation of cysteines, and variable modifications were set to oxidation of methionines, asparagine and glutamine deamidation, and protein N-terminal acetylation

Mechanisms of disease: mitochondrial dysfunction in sensory neuropathy and other complications in diabetes

The authors state, however, that this potential appears to be lower than the normal physiological levels of GSH